Abstract

Summary

This thesis demonstrates that a high-fat, high-sugar diet disrupts circadian clock gene expression (PER2, Per2) in food-reward brain regions including the Lateral Habenula and Nucleus Accumbens, altering day-night feeding patterns. For lighting and wellbeing practitioners, these findings highlight that diet-induced circadian disruption in reward circuits may compound or interact with light-driven circadian misalignment, suggesting that circadian health interventions should consider both photic and non-photic factors.
Abstract

Key Findings

  • fcHFHS diet altered day-night eating patterns and PER2 clock-protein expression in the Lateral Habenula in mice
  • Per2 gene expression was disrupted in the Nucleus Accumbens of rats on fcHFHS diet, even without changes in LHb clock genes
  • Pharmacological blockade of glutamatergic functioning in the LHb altered food intake in a time-dependent manner in both chow and fcHFHS-fed rats
  • Npas2 mutant and wild-type mice showed similar diet-induced disruptions in eating patterns, suggesting Npas2 is not a key mediator of fcHFHS-induced rhythm disruption
Categories

Categories

Sleep & Circadian Health: Examines how palatable diet disrupts circadian clock gene expression (PER2, Per2, Npas2) and 24-hour behavioral rhythms in reward-related brain areas.
The Science of Light: Investigates clock gene mechanisms and circadian pacemaking at the molecular level, relevant to understanding how non-photic zeitgebers like food interact with circadian systems.
Authors

Author(s)

ASB Velazquez
Publication Date

Publication Year

2018
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