Summary
This paper discusses the role of transcription factors Tbr2 and Isl2 in the development, maintenance, and survival of retinal ganglion cells (RGCs), which are crucial for vision.
Categories
Eye health: The paper explores the role of transcription factors in the development and survival of retinal ganglion cells, which are crucial for vision.
Education and learning: The paper provides new insights into the role of transcription factors in retinal ganglion cell development and survival, contributing to the broader understanding of vision neuroscience.
Aging: The paper indirectly discusses aging as it mentions the increase in age-related forms of blindness due to the inability of neurons in the retina to regenerate.
Author(s)
S Abed
Publication Year
2023
Related Publications
Eye health
- Phototransduction by retinal ganglion cells that set the circadian clock
- Color appearance models
- Diminished pupillary light reflex at high irradiances in melanopsin-knockout mice
- Strange vision: ganglion cells as circadian photoreceptors
- Genetic reactivation of cone photoreceptors restores visual responses in retinitis pigmentosa
Education and learning
- Color appearance models
- Genetic dissection of retinal inputs to brainstem nuclei controlling image stabilization
- The role of the circadian system in the etiology and pathophysiology of ADHD: time to redefine ADHD?
- How to report light exposure in human chronobiology and sleep research experiments
- Simulation-aided occupant-centric building design: A critical review of tools, methods, and applications
Aging
- Light therapy and Alzheimer's disease and related dementia: past, present, and future
- Function of human pluripotent stem cell-derived photoreceptor progenitors in blind mice
- Melanopsin-expressing intrinsically photosensitive retinal ganglion cells in retinal disease
- Neuroprotective strategies for retinal ganglion cell degeneration: current status and challenges ahead
- Combinatorial effects of alpha-and gamma-protocadherins on neuronal survival and dendritic self-avoidance