Abstract

Summary

This thesis investigates how acute lipopolysaccharide (LPS) administration disrupts circadian rhythm regulation by modifying intracellular signaling pathways in the suprachiasmatic nuclei (SCN) and pineal gland, including phosphorylation of GSK3β, ERK1/2, and STAT3. For lighting and healthcare applications, these findings suggest that immune challenges (e.g., infection, systemic inflammation) can interfere with the biological clock's responsiveness, potentially informing light therapy protocols for patients experiencing illness-related circadian disruption.
Abstract

Key Findings

  • LPS administration substantially modified levels of phosphorylated GSK3β, ERK1/2, and STAT3 in the SCN and pineal gland, indicating immune-driven disruption of core circadian signaling.
  • Effects were particularly pronounced in the pineal gland, suggesting that immune activation may suppress or alter melatonin synthesis pathways.
  • LPS caused phase shifts in circadian rhythmicity and behavioral changes, consistent with immune-circadian crosstalk mediated through the SCN.
Categories

Categories

Sleep & Circadian Health: Examines how endotoxin (LPS) disrupts circadian rhythmicity, melatonin synthesis, and phase shifts in the suprachiasmatic nuclei and pineal gland.
The Science of Light: Investigates cellular signaling cascades (GSK3β, ERK1/2, STAT3) in the SCN and pineal gland that underpin circadian entrainment and melatonin regulation.
Authors

Author(s)

P Štěrbová
Publication Date

Publication Year

2015
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